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Ruiyu Jia

Publications and source records attributed to Ruiyu Jia.

2 recordsLinked to original sources

Activation-Flexible ANN-to-SNN Conversion with Finite-State Markov Neurons

Most ANN-to-SNN conversion methods rely on a specific correspondence between the source activation and the spiking neuron dynamics. We propose a finite-state continuous-time Markov chain (CTMC) neuron framework whose stationary spike flux can approximate every continuous nonnegative monotone activation function on a compact interval. For a generalized CTMC family with affine input-dependent transitions, we prove uniform approximation to arbitrary accuracy over this function class and derive an explicit approximation error bound. In practice, two- and three-state CTMCs fit ReLU, sigmoid, softplus, and clipped ReLU on the evaluated input ranges, and we evaluate corresponding MLP conversions for each activation with layerwise rate scaling. Moderate clipping improves the conversion cost-accuracy tradeoff on the MNIST MLP and reduces SynOps by 27% on VGG-11/MNIST at matched ANN-SNN accuracy gap criteria, whereas the trend reverses on VGG-11/CIFAR-10. Mean-field and layerwise diagnostics indicate that finite-window sampling and terminal-layer mismatch are the main residual errors. Overall, our results establish finite-state CTMC neurons as a theoretically grounded framework for activation-flexible ANN-to-SNN conversion beyond fixed activation-neuron correspondences.

cs.NE↗

Revealing Mammographic Phenotypes in Deep Learning Breast Cancer Risk Models

Mammogram-based deep learning models have improved breast cancer risk prediction, but the learned imaging patterns remain underexplored. Existing interpretability methods rely on single-image saliency maps, failing to identify recurring mammographic phenotypes across large patient cohorts. By clustering patch embeddings from a pre-trained model, Mirai, we isolate recurring phenotypes linked to 5-year cancer risk. Analyses show risk-increasing phenotypes capture complex structures (e.g., dense tissue, microcalcifications) and shortcut artifacts (e.g., clips). These phenotypes correlate strongly with older age and higher BI-RADS density. Our framework connects tissue patterns to AI risk scores, revealing clinical signatures and potential latent model confounders.

eess.IV↗